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anti epor rabbit polyclonal  (Bioss)


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    Structured Review

    Bioss anti epor rabbit polyclonal

    Anti Epor Rabbit Polyclonal, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 12 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti epor rabbit polyclonal/product/Bioss
    Average 94 stars, based on 12 article reviews
    anti epor rabbit polyclonal - by Bioz Stars, 2026-02
    94/100 stars

    Images

    1) Product Images from "Differential regulation by CD47 and thrombospondin-1 of extramedullary erythropoiesis in mouse spleen"

    Article Title: Differential regulation by CD47 and thrombospondin-1 of extramedullary erythropoiesis in mouse spleen

    Journal: eLife

    doi: 10.7554/eLife.92679


    Figure Legend Snippet:

    Techniques Used: Cell Isolation, Recombinant, Control



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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    <t>EPOR-ERK1</t> signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.
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    Image Search Results


    Journal: eLife

    Article Title: Differential regulation by CD47 and thrombospondin-1 of extramedullary erythropoiesis in mouse spleen

    doi: 10.7554/eLife.92679

    Figure Lengend Snippet:

    Article Snippet: Antibody , Anti-EPOR (Rabbit polyclonal) , Bioss; Thermo Fisher , Cat#: BS-1424R , FACS (1 μg/100 μl/1 million cells).

    Techniques: Cell Isolation, Recombinant, Control

    EPOR-ERK1 signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.

    Journal: Journal of Neuropathology and Experimental Neurology

    Article Title: Angiogenic responses are enhanced by recombinant human erythropoietin in a model of periventricular white matter damage of neonatal rats through EPOR-ERK1 signaling

    doi: 10.1093/jnen/nlae001

    Figure Lengend Snippet: EPOR-ERK1 signaling pathway proteins. Bar graph and representative bands depicting protein expression 15 minutes (A) and 30 minutes (B) after hypoxic/ischemic-induced brain injury. EPO treatment increases expression of EPOR and p-ERK1 proteins. The p42/44 MAP (ERK1/2) kinase inhibitor, U0126, downregulated p-ERK1 expression, while EPOR expression was unchanged. There were no changes in ERK protein expression among the groups. *p < 0.05; NS, no significant difference.

    Article Snippet: The membranes were then incubated with primary polyclonal antibodies recognizing EPOR (1:1000, FNab02816, Wuhan Fine Biotech, Wuhan, China), p-ERK1 (1:500, ab131438, Abcam, Cambridge, United Kingdom), ERK (1:1000, ab115799, Abcam), and CD34 (1:1000, AF-4117, R&D Systems, Minneapolis, MN) at 4°C overnight.

    Techniques: Expressing

    CD34 + cells/CD34 protein expression was enhanced by active EPOR-ERK1 signaling after hypoxic/ischemic-induced brain injury. (A, B) Immunostaining and quantification of CD34 + cells in rat pups brain. EPO augmented the number of CD34 + cells, while the number was decreased following inhibition of p-ERK1 by U0126. (C) Bar graph and representative western blot images of CD34 protein expression. CD34 protein was upregulated in response to EPO treatment and downregulated following inhibition of p-ERK1 by U0126. *p < 0.05; NS, no significant difference. Scale bar: 100 µm.

    Journal: Journal of Neuropathology and Experimental Neurology

    Article Title: Angiogenic responses are enhanced by recombinant human erythropoietin in a model of periventricular white matter damage of neonatal rats through EPOR-ERK1 signaling

    doi: 10.1093/jnen/nlae001

    Figure Lengend Snippet: CD34 + cells/CD34 protein expression was enhanced by active EPOR-ERK1 signaling after hypoxic/ischemic-induced brain injury. (A, B) Immunostaining and quantification of CD34 + cells in rat pups brain. EPO augmented the number of CD34 + cells, while the number was decreased following inhibition of p-ERK1 by U0126. (C) Bar graph and representative western blot images of CD34 protein expression. CD34 protein was upregulated in response to EPO treatment and downregulated following inhibition of p-ERK1 by U0126. *p < 0.05; NS, no significant difference. Scale bar: 100 µm.

    Article Snippet: The membranes were then incubated with primary polyclonal antibodies recognizing EPOR (1:1000, FNab02816, Wuhan Fine Biotech, Wuhan, China), p-ERK1 (1:500, ab131438, Abcam, Cambridge, United Kingdom), ERK (1:1000, ab115799, Abcam), and CD34 (1:1000, AF-4117, R&D Systems, Minneapolis, MN) at 4°C overnight.

    Techniques: Expressing, Immunostaining, Inhibition, Western Blot

    Expression of angiogenic factors was increased by active EPOR-ERK1 signaling after hypoxic/ischemic-induced brain injury. EPO increased the levels of angiogenic factors ( VEGF and Ang-1 ), while the levels were decreased following inhibition of p-ERK1 by U0126. *p < 0.05.

    Journal: Journal of Neuropathology and Experimental Neurology

    Article Title: Angiogenic responses are enhanced by recombinant human erythropoietin in a model of periventricular white matter damage of neonatal rats through EPOR-ERK1 signaling

    doi: 10.1093/jnen/nlae001

    Figure Lengend Snippet: Expression of angiogenic factors was increased by active EPOR-ERK1 signaling after hypoxic/ischemic-induced brain injury. EPO increased the levels of angiogenic factors ( VEGF and Ang-1 ), while the levels were decreased following inhibition of p-ERK1 by U0126. *p < 0.05.

    Article Snippet: The membranes were then incubated with primary polyclonal antibodies recognizing EPOR (1:1000, FNab02816, Wuhan Fine Biotech, Wuhan, China), p-ERK1 (1:500, ab131438, Abcam, Cambridge, United Kingdom), ERK (1:1000, ab115799, Abcam), and CD34 (1:1000, AF-4117, R&D Systems, Minneapolis, MN) at 4°C overnight.

    Techniques: Expressing, Inhibition